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a drug decreases heart rate by blocking a receptor on cardiac pacemaker cells. this drug probably binds to receptors is beta-1 adrenergic.
Heart rate and myocardial the cardiac pacemaker contractility are regulated by beta-1-adrenergic receptors, but during stress and when epinephrine release is provoked, cardiac beta-2 receptors are stimulated, which results in further increases in heart rate and contractility.The G protein-coupled receptors known as beta(1)- and beta(2)-adrenergic receptors are found all over the body and act as receptors for the catecholamines epinephrine and norepinephrine. In the therapy of asthma and heart failure, they serve as targets for therapeutic agonists and/or antagonists.A class of medications known as beta1-adrenergic agonists, commonly referred to as beta1-adrenergic receptor agonists, binds specifically to the beta-1 adrenergic receptor. They consequently affect the heart more selectively.Sinoatrial (SA) nodal, atrioventricular (AV) nodal, and ventricular muscle firing are all increased with targeted activation of the beta-1 receptor in the heart, raising heart rate and contractility. The stroke volume and cardiac output will rise along with these two elevated values.
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